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In this paper, we explore the potential role of certain amyloidogenic proteins, including amylin, amyloid beta, the prion protein, huntingtin, and -synuclein, in disease processes that result in the accumulation of deposits of protein fibrils, along with lipid membrane components of damaged mitochondria, which we argue may be a mechanism to sequester deuterium in order to reduce the deuterium burden in the tissues

Benedict Jephcote, Editor of Diabetes.co.uk, said: It is encouraging to see that researchers are looking at other ways of stimulating delivery of GLP-1 medication, which has typically been delivered via injection

doi: 10.1016/j.freeradbiomed.2020.09.015 66 MengesPKesslerWKloeckerCFeuerherdMGaubertSDiedrichSet al

It also acts on appetite/satiety centres, contributing to reduced food intake

HERA Adherent HMEC-1-FcRn cells 69 provided by Dr