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J Clin Invest 134(17) Ghosal S et al (2017) Disruption of glucagon-like peptide 1 signaling in Sim1 neurons reduces physiological and behavioral reactivity to acute and chronic stress

Acetylcholine acts through nicotinic receptors to enhance the firing rate of a subset of hypocretin neurons in the mouse hypothalamus through distinct presynaptic and postsynaptic mechanisms

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Membrane potential values were not compensated to account for junction potential (8 mV)